Central Actions of Angiotensin and Related Hormones

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Pathologic Basis of Disease 8th ed. Saunders Elsevier. Neurosurg Clin N Am. Merck Manual Home Edition. Medscape Cardiology. Pharmacological Reviews.

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Endocrine Reviews. Clinical Science. Current Opinion in Nephrology and Hypertension. Current Hypertension Reports. Nature Reviews.

Part I - Regulation of Blood Pressure (Hormones)

Res Exp Med Berl. Physiology of the cardiovascular system.

Central venous Right atrial ventricular pulmonary artery wedge Left atrial ventricular Aortic. Ventricular remodeling. Compliance Vascular resistance Pulse Perfusion. Pulse pressure Systolic Diastolic Mean arterial pressure Jugular venous pressure Portal venous pressure. Baroreflex Kinin—kallikrein system Renin—angiotensin system Vasoconstrictors Vasodilators Autoregulation Myogenic mechanism Tubuloglomerular feedback Cerebral autoregulation Paraganglia Aortic body Carotid body Glomus cell.

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Physiology of the kidneys and acid-base physiology. Solvent drag sodium chloride urea glucose oligopeptides protein.

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Renal blood flow Ultrafiltration Countercurrent exchange Filtration fraction. Human homeostasis. Predictive Thermoregulation. Angiotensin receptor modulators.

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Categories : Human homeostasis Cardiovascular physiology Endocrinology. Hidden categories: Webarchive template wayback links Use dmy dates from May All articles with unsourced statements Articles with unsourced statements from March Namespaces Article Talk. Angiotensin, specifically angiotensin II, binds to many receptors in the body to affect several systems. It can increase blood pressure by constricting the blood vessels. It can also trigger thirst or the desire for salt.

In the adrenal glands, angiotensin stimulates aldosterone production. This hormone causes the body to retain sodium. In the kidneys, sodium retention triggered by angiotensin changes the way the blood is filtered, causing increased water re-absorption to increase the volume of blood. This, again, increases blood pressure.


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While angiotensin has a complex series of effects on the body, the primary results are higher blood volume, blood pressure and sodium content. People can struggle with angiotensin in one of two ways: Too much angiotensin can cause the body to retain too much fluid or to have elevated blood pressure levels not caused by other problems. High angiotensin levels can also cause the heart to grow, leading to heart failure.

Angiotensin blockers can help in these situations by blocking the receptor sites that take up angiotensin, but this can lead to too much potassium retention. Working with an endocrinologist is important to ensure the body remains healthy while balancing angiotensin levels.


  1. Alternative names for angiotensin.
  2. Renin–angiotensin system.
  3. Motivation and Emotion (Routledge Modular Psychology).
  4. Low angiotensin levels can also be problematic. Angiotensin deficiency can prevent the regulation of blood volume and pressure, increase retention of potassium, and lead to a loss of sodium and more urine output, as the body releases needed fluid. This causes a lower blood pressure. Angiotensin is essential to regulating blood pressure.

    Central actions of angiotensin and related hormones

    If you are struggling with maintaining a healthy blood pressure, consider asking your doctor:. If you are not working with an endocrinologist to help manage angiotensin levels, you need to find one in your area. An endocrinologist has the knowledge and understanding to help keep angiotensin levels healthy. To take the next step, find a specialist near you.

    More in this section What is Endocrinology? Keep Your Body In Balance! The Hormone Health Network is the public education affiliate of the Endocrine Society dedicated to helping both patients and doctors find information on the prevention, treatment and cure of hormone-related conditions.

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    Visit the Endocrine Society. All Network materials, including the content on this site, are reviewed by experts in the field of endocrinology to ensure the most balanced, accurate, and relevant information available. The information on this site and Network publications do not replace the advice of a trained healthcare provider.

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